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ibogaine.co.uk An Introduction
to Ibogaine
An Introduction to Ibogaine
by Nick Sandberg
ISBN 0-9538348-1-6
www.ibogaine.co.ukThis piece is not subject to copyright and may be reproduced
Written in 2001, and occasionally updated
Chapters
Introduction
Ibogaine
The Problems of Developing Ibogaine
Casual Ibogaine Treatment
Ibogaine Treatment
How Ibogaine Works
The Bwiti
Ibogaine for Self-development
Iboga Visions
Bibliography
Introduction
Ibogaine is a psychoactive indole alkaloid derived from the rootbark of an African plant - Tabernanthe iboga. In recent years it has been increasingly noted for its ability to treat both drug and alcohol addiction. Both scientific studies and widespread anecdotal reports appear to suggest that a single administration of ibogaine has the ability to both remove the symptoms of drug withdrawal and reduce drug-craving for a period of time after administration. In addition, the drug's psychoactive properties (in large doses it can induce a dreamlike state for a period of hours) have been widely credited with helping users understand and reverse their drug-using behaviour.
Studies suggest that ibogaine has considerable potential in the treatment of addiction to heroin, cocaine, crack cocaine, methadone, and alcohol, with some suggestion that it further be useful in treating tobacco dependence. It has also been suggested that the drug may have considerable potential in the field of psychotherapy, particularly as a treatment for the effects of trauma or conditioning.
A single administration of ibogaine typically has three effects useful in the treatment of drug dependence. Firstly, it causes a massive reduction in the symptoms of drug withdrawal, allowing relatively painless detoxification. Secondly, many users report, and scientific studies confirm, a marked lowering in the desire to use drugs is experienced for a period of time after taking ibogaine, typically between one week and several months. Finally, the drug's psychoactive nature is reported to help many users understand and resolve the issues behind their addictive behaviour.
Ibogaine can be easily administered, in capsule form, and has no addictive effects itself. It is essentially a "one-shot" medication and, used in a clinical setting with proper client screening procedures, the drug thus far appears to be safe to use. Whilst it is rare for an individual to stop using drugs permanently from a single dose of ibogaine, as the initial component in an overall rehabilitation programme the drug would appear to offer much potential.
Although approved for clinical trials (trials on humans) for the treatment of addiction in the US in the early 1990s, problems with financial backing have so hindered the development of ibogaine that, as of mid 2001, it remains undeveloped and thus unavailable to the majority of addicts worldwide. There are however a couple of private clinics, located around the Caribbean and in Mexico, that offer ibogaine treatment at prices starting around £4,000, and some lay treatment providers offer lower cost treatment, without medical facilities, in Europe. In addition, ibogaine, either in pure form or as a plant extract, has become available from some lay sources on the internet.
Ibogaine's current legal status in the UK, and much of the rest of the world, is that of an unlicensed, experimental medication, and it not therefore an offence to possess the drug, though to act as a distributor may be breaking the law. Ibogaine is a restricted substance (possession is illegal) in some countries, including the US, Switzerland, Sweden and Belgium.
Ibogaine
Of the various substances that have, at one time or another, been proposed as being useful in the treatment of drug or alcohol dependence, ibogaine would certainly appear to be the one offering the greatest real potential. A slightly psychoactive indole alkaloid derived from an African plant, the drug, in plant form, has been used by indigenous groups for millenia. The Bwiti, a Central African religious group, use the rootbark of the Tabernanthe iboga plant for a variety of social and religious purposes, most notably as the central component of a "rite of passage" initiation ceremony intended to confer the status of adulthood upon new group members. In the West, ibogaine is usually administered in the form of the hydrochloride - a fine off-white powder either lab synthesized or chemically extracted from the rootbark.
When administered to persons seeking to beat addiction to heroin, methadone, cocaine or alcohol, a single dose of ibogaine typically achieves the following. Firstly, the complete removal or severe attenuation of the symptoms of drug withdrawal, allowing painless detoxification (occurs with approx. 90% of subjects). Secondly, the removal of the desire to use drugs for a period of between one week and three months (occurs with approx. 60% of subjects). Finally, the revealing of personal issues underlying drug-using behaviour, leading to long-term drug-abstinence (occurs with approx. 30% of subjects).
Ibogaine is not itself addictive and the drug may be taken a second time to help preserve a drug-free state. It should be noted, however, that relatively few people permanently beat addiction solely through using ibogaine, and the treatment should thus be regarded as simply an initial component in an overall rehabilitation strategy.
The discovery that ibogaine could treat drug addiction is usually credited to Howard S. Lotsof - a New York based former heroin user who first took ibogaine in 1962. Lotsof took ibogaine believing it to be a new recreational drug but, 30 hours later, suddenly realized he wasn't experiencing heroin withdrawal, and had no desire to seek drugs. Subsequent casual experimentation by addict friends revealed that this effect was common to others.
Some 20 years later, Lotsof returned to his discovery and set about trying to bring it to the market. He initially set up a charitable foundation with the aim of promoting and developing ibogaine as an anti-addiction medication but, dismayed by the lack of interest shown, later decided to form a company, NDA International, believing a business concern would more likely attract the necessary financial backing. NDA filed patents for the use of ibogaine in the treatment of addiction and began to carry out treatments to better evaluate the drug's potential.
Because, by this time, ibogaine had been made a Schedule 1 restricted substance in the USA (ibogaine was banned along with LSD and psilocybin in the early seventies) NDA chose to carry out experimental ibogaine treatments in Holland. Jan Bastiaans, a highly-regarded Dutch psychotherapist, partnered him and, over the early years of the nineties, they treated some 30 addict volunteers, the results of which were later medically assessed by Dr Ken Alper in a scientific paper (see How Ibogaine Works for ref).
The nineties, after a promising start, proved to be a tough time for ibogaine. In 1991, the US National Institute for Drug Abuse (NIDA), impressed by case reports and animal studies, began studying ibogaine with a view to evaluating its safety. They constructed protocols for the treatment of addiction. In 1993, the US Food and Drug Administration (FDA), who oversee the development of new drugs, approved clinical trials with ibogaine, to be carried out by Dr Deborah Mash of the University of Miami School of Medicine, on behalf of Howard Lotsof's corporation, NDA International.
It was at this point that things started to go astray. The death of a young female heroin addict during treatment in Holland brought an abrupt end to the Dutch project. A subsequent inquest did not find the project organizers guilty of negligence but the lack of scientific knowledge about the effects of ibogaine hindered the establishing of an actual cause of death. (It was believed that the surreptitious smoking of opiates during treatment may have been responsible).
The approved clinical trials commenced but contractual and funding problems that arose between NDA International and the University of Miami brought the trials to a close before completion, (the drug's safety was not an issue). A lengthy legal battle between the two ensued, and developmental work came to a standstill.
In March 1995, after several years spent progressively becoming more interested in ibogaine, a review committee at NIDA suddenly decided to greatly reduce further activity with the drug, apparently having been influenced by critical opinions from the pharmaceuticals industry. Officially, it was reported that the death in Holland was of concern, and that NIDA were disappointed that ibogaine was only shown to keep people off drugs for a period of months, not forever. Howard Lotsof has subsequently pointed out that the death, whilst tragic, was likely caused by concurrent opiate usage and, with regard to the second point, that any drug that could put, say, cancer or AIDS into complete remission for a period of months would be being developed as a matter of national urgency.
Over the last five years, very little has happened. The escalating legal battle between NDA International and the University of Miami, each suing the other for alleged breaches of contract, appears to have ended with the bankruptcy of the former. Yet, as of mid 2001, the precise outcome is not clearly established.
Meanwhile, widening knowledge of the effects of ibogaine has resulted in casual treatments being provided by various individuals in different countries. Though usually undertaken with good intentions, these treatments have frequently been carried out by people with little medical knowledge, and this may have resulted in further tragic incidents.
A couple of small countries, notably Panama and St Kitts, have made ibogaine treatment legally available at private clinics, but only at prices starting around UK£7,000 per treatment (approx US$10,000. Unlicensed medical clinics in Mexico currently offer the treatment slightly more cheaply). As of mid 2001, ibogaine remains in a legal nowhere-land, desperately needed by millions of addicts worldwide, but, tragically, little closer to becoming easily available.
The Problems of Developing Ibogaine
Ibogaine development has been beset with hold-ups for years. The existing legal disputes may now be close to resolution, but ibogaine still needs the participation of a pharmaceutical company for it to make it to the mass market. The business of developing new medications is solely in the hands of the private sector - the pharmaceutical corporations - and the problems that drugs companies appear to have with ibogaine are many.
Firstly, as a drug derived from a natural source, patent options are more limited than they would be for a drug that can only be created in the lab. Potentially, this greatly reduces the level of financial return that the drug could provide, of serious concern considering the degree of backing needed to bring a new drug to the market. Whilst, in the West, there are governmental provisions in place to encourage companies to develop drugs that could be socially useful, to date no one seems interested in taking advantage of them for ibogaine.
Secondly, ibogaine is not a maintenance drug - it is not taken repeatedly over a short period of time - and is usually administered only once. As a general rule, medications developed by the drugs companies, for whatever purpose, are maintenance drugs, for only maintenance drugs allow sufficient financial return to justify the necessary prior outlay on research and development.
Thirdly, industry insiders relate that there are public relations concerns when developing medications for groups that are negatively socially marginalized in the way drug addicts have become. Drug companies, like most modern corporations, are acutely image-sensitive and there are thus concerns that developing medications for addicts could bring about a deterioration in their overall market value.
Finally, some believe that bringing an addiction medication of ibogaine's potential to the market may present "conflict of interest" problems, of dubious moral worth, to other corporate bodies involved with the sale of licensed recreational substances such as alcohol or tobacco.
The root of the problem that ibogaine faces in becoming available is that our society lacks any mechanism by which a substance of this nature, offering high social benefits but only marginal direct financial return, can be developed. Drugs companies are shareholder based, and so can only develop medications that offer sustained, direct financial return. Whilst ibogaine potentially offers immense savings to government in terms of reduced spending on social welfare and crime prevention, there is no mechanism by which this saving at a public level can be used to induce a corporation to develop the drug.
Assuming the absence of corporate backing, about the most likely route by which ibogaine might become legally available is via projects carried out by local government drug dependency units. Projects of this nature, once started, would allow addicts access to safe, low-cost treatment and, as each project generated more knowledge and data, so drug treatment centres in other areas could make use of the same to develop their own ibogaine protocols. As of mid 2001, however, no projects of this nature are underway, although East European countries appear to be at the forefront of those interested. In addition, the medical laws of some countries allow registered practitioners to prescribe an unlicensed medication like ibogaine, usually providing the subject has given their "fully-informed consent."
Casual Ibogaine Treatment
With ibogaine treatment now more available than ever before, in an ever-widening range of settings, more and more knowledge about the drug is gathering. At the time of writing, March 2007, one thing that is becoming increasingly clear is that there is a reasonable degree of risk associated with taking the drug. At least 12 people are recorded as having died in connection with taking ibogaine or other iboga substances over the last decade or so, and there is reason to believe that the number may be higher, with other deaths having occurred in non-clinical settings and without being recorded.
Here is some safety-related information about the drug:
- There is an inherent level of risk with ibogaine treatment. Twelve people are known to have died in connection with taking ibogaine or other iboga alkaloids. In actuality, the figure is likely higher, given that ibogaine is frequently administered in surroundings where people may be reluctant to contact the authorities in the event of something going wrong. Statistically, a ballpark figure for deaths during treatment is probably of the order of 1 in 300. (This is based on 12 recorded deaths having occurred within 3611 recorded treatments, outside of Africa, as of March 2007). The following factors have been identified as having caused death:
* having a pre-existing heart condition, sometimes one not detectable by EKG
* using opiates when on ibogaine, or shortly afterwards
* using the rootbark or iboga extract. Ibogaine HCl is statistically much safer
* taking ibogaine outside of a clinical facility. Persons taking ibogaine need constant supervision and, ideally, online heart monitoring
- Ibogaine is principally recognised for its ability to vastly reduce the symptoms of drug withdrawal, thus allowing addicts to detox relatively painlessly. Any other claims made for the drug, such as that it creates long-term drug-abstinence, or removes the effects of trauma or conditioning in either addicts or non-addicts, may have a degree of truth but are a great deal less substantiated.
- You must be medically tested before you take ibogaine. Proper clinical testing of heart and liver function are the absolute minimum. The site author is not aware of any reputable treatment provider who would allow you to take ibogaine without prior medical testing. Do not go with someone who does not insist on it. Ideally, you should have constant monitoring of heart function whilst on the drug, and medically-trained staff present.
- Beware of listening excessively to the advice of just one individual when deciding whether or not to take ibogaine. Ibogaine's effects can be life-changing, and it is common for someone who has had a very positive experience to do their utmost to "spread the message," possibly allowing their enthusiasm to override the very real concerns about safety.
- If you are thinking of taking ibogaine for personal development and haven't yet been involved in proper therapy (therapy where there's an open admission by the individual of the presence of emotional issues), be aware that you may be being attracted to a "quick fix" strategy that avoids really dealing with deeper issues. If this is the case, ibogaine could possibly make things worse. For some, using psychoactive substances can invoke disturbing reactions as the mind's defences struggle to keep down rising repressed material. Drugs like ibogaine, ketamine, LSD and MDMA (Ecstasy), have been used in the past by therapists, but only as one component of an overall therapeutic strategy. Using the drug out of this context could cause more harm than good.
Ibogaine Treatment
(This article has been reproduced for interest value only).
Ibogaine, an indole alkaloid derived from an African plant source, has for many years been recognized for its ability to interrupt drug dependency. Specifically, it can be effective in the treatment of withdrawal from heroin, methadone, cocaine (inc. crack cocaine), amphetamine, and alcohol.
Although it is slightly psychoactive, ibogaine should not be confused with drugs like LSD or psilocybin. Ibogaine's effects are far longer lasting and can be intensely physical in some users. The drug should be treated with respect and not administered by persons unfamiliar with basic medical procedures. Because vomiting can be a problem with ibogaine treatment, persons administering should ensure especially that they are fully familiar with resuscitation procedures and have rapid access to the emergency services should they be required. It is important persons interested in receiving ibogaine treatment are properly screened. Failure to do so may have resulted in previous tragic accidents. Heart (EKG) and liver (Blood) screening are the absolute minimum.
PREPARATION OF THE CLIENT - The prospective client should attend several informal interviews to ensure he or she is fully aware of the following information relating to ibogaine treatment:
(i) - that ibogaine is principally a detox tool and that, whilst it can help with drug-craving for brief periods as well as help a person understand why they started using drugs, it will still be up to them to stay off. As a general rule, addicts who regard ibogaine as simply something which is supposed to "cure them" rarely have success.
(ii) - that ibogaine is an experimental medication, not recognized as a licensed medicine anywhere in the Western world, and that other options for treating their addiction exist.
(iii) - that deaths have occurred in association with ibogaine treatment, and that it must therefore be regarded as having a definite level of risk, though proper client screening procedures should be able to keep this to a minimum. Specifically, anyone with any history of heart problems should be very wary of taking ibogaine. In recent years there have been several reports of mysterious deaths associated with cardiac problems.
A basic level of physical and psychological screening is essential prior to a person being considered suitable for ibogaine treatment. A blood test should be undertaken to check for liver abnormalities and to ensure general health is good. An EKG should be undertaken to check heart function. Problems with the liver, heart or lungs should result in exclusion from treatment unless subsequent professional medical opinion advises to the contrary. Many long-term addicts may have developed medical health problems which would make ibogaine treatment in a non-clinical setting dangerous. These tests can be often be organized by drug dependency units or private doctors.
Attention should also be paid to the clients' mental state. Persons exhibiting signs of significant mental disorder should be excluded from treatment.
DOSAGE - Assuming the client is sufficiently well to be treated, their bodyweight in kilos should be measured, and a suitable dose of ibogaine calculated.
Pure ibogaine HCl is typically administered at doses of around 10 milligrams per kilo bodyweight (mg/k) for men, and 9 mg/k for women. To calculate the dose, multiply the client's bodyweight in kilos by either 10 (for men) or 9 (for women) and you will have the dose in milligrams.
Example: An 8 stone female alcoholic will require about 460mgs of ibogaine HCl, a little under half a gram. (8 stone x 14 = 112 lbs. 112 / 2.2 = 50.9 kgs. 50.9 x 9 = 458mgs)
Note that this is for pure ibogaine HCl, one of two forms of the drug commonly available in Europe. The other is the "Indra iboga extract," which is believed to be approximately one quarter the strength of pure HCl, meaning clients will require roughly four times the amount. Although the "Indra" product is becoming increasingly available in Europe, it is known to induce more vomiting than the HCl. In January 2000, a 40 year old heroin addict died in London after vomit clogged his airways some 40 hours after taking a dose of this extract.
For opiate addicts, such as those using heroin or methadone, the dose of ibogaine HCl is typically doubled, to around 20mg/k for men, and 18mg/k for women. This is because the opiates in a person's system partially block ibogaine's effect.
It is recommended that ibogaine only be given as a single dose, in the range of 9-10 mg/k. From what is known, this appears to be the safest way to take the drug, bearing in mind that higher doses can always be taken in subsequent sessions if necessary. When re-dosing, it is recommended to wait at least one month as ibogaine and its metabolites linger in the body.
TREATMENT PREPARATION - It is very important that the client's drug intake be regulated for 24 hours prior to taking the main dose of ibogaine. This will prevent the ibogaine from reacting with any other drugs still in the body, which research indicates may lead to adverse reactions. This means that no heroin, no cocaine and no other drugs should be taken for a minimum of 12 hours prior to taking the main dose of ibogaine. No methadone for a minimum of 24 hours. Drug use for the days prior to treatment should therefore be planned in advance to ensure this is possible. In addition, no stimulants should be taken for at least 24 hours prior to taking the main dose of ibogaine. Normal doses of benzodiazepines like valium can safely be taken prior to ibogaine to assist in reducing anxiety or to help the client sleep if necessary.
Ibogaine is recognized as having the ability to potentiate other drug reactions, meaning it is very important persons under its influence do not get access to drugs. Any level of opiate or cocaine usage whilst on ibogaine could be very dangerous.
24 hours prior to taking the main dose of ibogaine, a test dose of about 100mg of the drug should be taken. Allergic reactions have not been reported to the best of the writer's knowledge but, in the event of one occurring, the treatment should not proceed. Some minor level of ataxia, (difficulty in standing upright), nausea, and aural amplification may be experienced at this dose level. This is quite normal.
Food consumption should cease about 12 hours prior to the main dose of ibogaine being taken. To make this easy to bear, many people take ibogaine first thing in the morning, as a replacement for their morning fix. 1 hour prior to taking the main dose, an anti-nauseant such as domperidone (or similar travel sickness medication) may be taken to try and reduce nausea.
The treatment setting is important in that the client should feel relaxed and relatively easy in themselves. This will help to limit anxiety. Noise should be low throughout (ibogaine causes sounds to be heard much louder than usual), and the light level adjustable. Remember that ibogaine incapacitates some people for several days, so make sure that peaceful, dimly lit conditions can be maintained.
A "sitter" should be present with the client for the duration of the experience, which usually lasts between 20 and 30 hours, but in some cases has been known to go on for 3 days. This should ideally be someone experienced in ibogaine administration, or otherwise a close friend. It is unlikely much communication will be attempted in this time and the client should therefore be attended in peace. Requests for water may be fulfilled but nothing else should be taken.
THE EXPERIENCE - The client will likely experience the drug taking effect after between 30 minutes and 2 hours. Withdrawal symptoms should be eliminated or easily manageable. There will likely be ataxia (problems getting upright) accompanied by a buzzing noise in the ears. Sounds will become louder, bright light hard to bear. Some people report feeling nauseous and there may be a sensation of pulsing in the body, rather as though it were being "cranked up to a new frequency." These sensations are quite normal.
Vomiting within 3 hours of taking the main dose may result in some of the ibogaine leaving the body before it can be absorbed. In such circumstances, giving more may be considered or perhaps the treatment aborted. Examining the vomit may reveal if the drug has left the body. Be aware of the dangers of both overdosing and using stepped doses if considering giving more ibogaine to make up for that lost in vomit, especially if this is the first time someone has used the drug.
The experience of taking ibogaine varies so much from person to person, it is difficult to prejudge just what will happen for any one individual. However, there are generally two, distinct phases to the experience.
First, the "oneirophrenic" or "dream-creating" phase. This generally lasts several hours and usually consists of the user experiencing dream-like visions with eyelids closed, which disappear once the eyes are open. The visions may appear to be actual memories running, rather as though a film of one's life was being shown inside the head, or may take the form of characters acting out roles, rather as though a play was taking place inside the head. However, many people report no visual sensations and this is not a problem. People may experience feelings and sensations associated with childhood and early life.
Secondly, the "processing" phase, which follows once the first stage is concluded. This phase is characterized by high levels of mental activity - interiorized processing that allows the material revealed in the first phase to be assimilated and interpreted. People frequently experience comprehending for the first time the reasons why they became involved with drugs. Though ibogaine affects different people in different ways, the oneirophrenic phase typically starts 1-2 hours after taking the main dose, and the processing phase about 3-6 hours later, usually lasting for between 8 and 14 hours. People sometimes experience very negative feelings on ibogaine. If this appears to be happening, the person attending could try to give them reassurance that things are OK. Whatever arises will pass.
What is described above is a typical session but it is by no means unknown for people to be up and moving around within a few hours of taking the main dose, apparently having experienced very little. Alternately, some remain in bed for half a week. In addition, opiate addicts frequently experience little or nothing of the "oneirophrenic" phase. Sessions that are over quickly are usually less effective, and ibogaine does appear to have very little effect on some individuals, regardless of dose level.
Potential treatment providers please note: It is important to realize just how variable the drug's effects can be on different people. Tragic incidents can occur if safety procedures become lax after a string of successful treatments. Because, when ibogaine works, its effect can seem quite miraculous, it is very easy for people who are not medically experienced to start to relax pre-treatment screening procedures in their keenness to treat people and this is dangerous.
POST IBOGAINE - If the treatment has been successful, the client should be clean having experienced little or no withdrawal. In addition, many experience no desire to use drugs for a period of weeks afterward. Furthermore, some users report gaining insights into their drug-using behaviour. As a general rule, ibogaine is most effective for older addicts, a casual study indicating that those over 35 have a far better chance of staying clean than those in their twenties.
In cases where the treatment has been successful, but the client begins to experience the desire to use drugs again after some weeks, repeat dosing with ibogaine can be undertaken. Remember that persons not currently using opiates require ibogaine at a maximum dose of around 10mg/k. Re-dosing with ibogaine at less than one month intervals may be risky, as metabolites of the drug can remain in the body for this length of time.
Melatonin and B vitamins have been suggested as useful after using ibogaine. Some believe they help sustain the drug's effect.
POST IBOGAINE REHAB AND THERAPY - A single dose or multiple doses, given over a period, of ibogaine will occasionally be enough to keep someone off drugs permanently. But for most the truth is that, unless suitable post-ibogaine work is undertaken, a fairly rapid relapse to old ways is likely.
It is simply not possible to give guidelines that will be valid for everyone, for we are all different. However, for many, the addict should ideally enter rehabilitation as soon as possible after the treatment. In the writer's opinion, the best rehab program, and likely the one most suitable for those who have just taken ibogaine, is the Residential Addiction Foundation (RAF) program run by the Humaniversity in Egmont-aan-Zee, Holland, see
www.humaniversity.nl for further details.
Other alternatives include any long-term (six months and up) residential rehab program available locally. Where residential rehab is not desirous, or not an option, suitable therapy should be seriously considered. Observations of the ethnic, religious use of the drug and first and second hand experience indicate to the writer that the most suitable types of therapy will be body-based and work around catharsis, confrontation and emotional release. "Talking only" type therapy, such as counselling may be effective in some cases but usually less so. Encounter therapy is often highly suitable for recovering addicts, as is primal therapy, bioenergetics, and indeed anything that sets out to assist the individual contact and release repressed emotions, frequently the root cause of addiction. More gentle, integrative work may also be useful. Dance structures such as 5 Rhythms or Biodanza may be helpful, either as a back-up to deeper work or on their own.
Attention should also be given to pleasure. Long term drug use will have likely had the effect of causing the addict's dopamine system to have been "hard-wired" to associate pleasure with drug use. This is the reason why many who have beaten addiction in the short term frequently relapse. A brief period of exposure to drug-using stimuli, especially at a time when a former addict feels vulnerable, often results in a return to addiction. Everyone needs pleasure and so the recovering addict must take steps to ensure they can get enjoyment out of life without using drugs. For the majority this will mean work on their sex lives. Sexual stimulation, and particularly orgasm, is the principle means by which the healthy body gains pleasure and releases tension. Work to increase the former user's ability to be intimate, both socially and sexually, is very important. Tantra workshops, touch therapy, or other intimacy-focussed processes are an excellent idea.
POST IBOGAINE PROBLEMS - Feelings of deep contentment - although less common with long term heroin users, many people using ibogaine feel in very high...
travinski is an alter ego and not a real person, i also like to play dress up when im not playing fictional chemist