http://www.scribd.com/do...bitory-and-Antioxidative

Results

An examination of the aqueous extracts of Banisteriopsis caapi cultivar Da Vine yielded two new alkaloidal glycosides, named banistenoside A (1) and banistenoside B (2), containing “azepino[1,2-a]tetrahydro-β-carboline” unique carbon framework. One additional new natural tetrahydronorharmine (4), four known β-carbolines harmol (3), tetrahydroharmine (5), harmaline (6) and harmine (7), two known proanthocyanidines (−)-epicatechin ( and (−)-procyanidin B2 (9), and a new disaccharide β-d-fructofuranosyl-(2 → 5)-fructopyranose (14) together with known sacharose (15) and β-d-glucose (16) were also isolated. In addition, the acetates of 1, 2, 8, 9, 14 and 15 (compounds 10–13, 17, 1 were also prepared. Harmaline (6) and harmine (7) showed potent in vitro inhibitory activity against recombinant human brain monoamine oxidase (MAO)-A and -B enzymes (IC50 2.5 and 2.0 nM, and 25 and 20 μM, respectively), and (−)-epicatechin ( and (−)-procyanidin B2 (9) showed potent antioxidant and moderate MAO-B inhibitory activities (IC50 < 0.13 and 0.57 μg/mL, and 65 and 35 μM). HPLC analysis revealed that most of the dominant chemical and bioactive markers (1, 2, 5, 7–9) were present in high concentrations in dried bark of large branch. Analysis of regular/commercial Banisteriopsis caapi dried stems showed a similar qualitative HPLC pattern, but relatively low content of dominant markers 1, 2, 7, and 9, which led to decreased MAO inhibitory and antioxidant potency.
Conclusion

Collectively, these results give additional basis to the existing claim of Banisteriopsis caapi stem extract for the treatment of Parkinsonism, including other neurodegenerative disorders.

that's the same action as rasagiline and l-deprenyl.
but an MAOI-B is not enough, it would need to be taken in conjunction with l-dopa. target area is the D1
receptors in the substantia nigra.

So caapi taken with mucuna tea or cowhedge would be effective then?

yes